Parchem Lab

Mechanisms of human development and disease

Why neural tubes fail to close.
Why placentas fail in preeclampsia.
How cancers reawaken the programs that built the embryo.
Why type 2 diabetes raises the risk of dementia.
Baylor College of Medicine & UTHealth Houston · Texas Medical Center
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A mother and developing fetus rendered in dots

Every person starts as one cell. That cell divides, and its descendants have to decide what to become: a neuron, a skin cell, part of a placenta. We study how those decisions get made, and what happens when they go wrong.

Getting it wrong early has consequences. If the tissue that forms the brain and spinal cord fails to close, a baby is born with a birth defect. If the placenta develops poorly, the pregnancy can turn dangerous for the mother. Cancer often works by switching these early programs back on in adult cells.

We work with mouse embryos, human stem cells, and tissue donated by patients. Following these questions has taken us into birth defects, pancreatic cancer, type 2 diabetes, and the complications of pregnancy.

We run the lab together. Ron is at Baylor College of Medicine. Jackie is at UTHealth Houston, where she also treats patients with high-risk pregnancies. Her patients are where a lot of our questions start.

Room N1020, Baylor College of Medicine · One Baylor Plaza, Houston, TX 77030
The Texas Medical Center is the largest medical complex in the world.
A mouse embryo inside its yolk sac, fluorescence microscopy
Mouse embryo across fluorescence channels, Parchem Lab
Mouse embryo with yolk sac and placenta, Parchem Lab
Pancreatic islet, fluorescence microscopy, Parchem Lab
published
What we've found

Developmental origins of birth defects

2023 – 2015
2023

miR-302 expands the developmental potential of cranial neural crest

Cranial neural crest cells pass through a transient window of open chromatin that permits both neuronal and ectomesenchymal fates. miR-302 holds that window open by targeting Sox9 to slow specification. Without it, cells commit early, accessibility narrows, and peripheral neurons are lost.

Keuls & Parchem, PNAS 2023
2020

A microRNA controls metabolism during neural tube closure

miR-302 directly represses three glycolytic enzymes in the closing neural tube. Loss upregulates glycolysis, shortens the cell cycle, and drives progenitor over-proliferation into cranial defects, confirmed by whole-embryo mass spectrometry. The first direct evidence linking a miRNA to metabolic control during neurulation.

Keuls et al., Int J Mol Sci 2020
2015

miR-302 is required for neural tube closure and embryonic viability

Deletion of miR-302a-d causes a fully penetrant anterior neural tube defect, with thickened neuroepithelium, increased progenitor proliferation, decreased apoptosis, and precocious neuronal differentiation. Compound mutants with miR-290 die before neurulation, revealing redundancy between the two clusters.

Parchem et al., Cell Reports 2015

Stem cell identity and reprogramming

2025 – 2013
2025

Reprogramming cannot proceed without these clusters

Returning to the question a decade later with genetic loss-of-function, we showed the miR-290 and miR-302 clusters are not merely correlated with reprogramming but required for it.

Ye et al., Stem Cells 2025
2016

One factor both builds and silences enhancers to set timing

Foxd3 simultaneously establishes and represses enhancers, using a single factor to hold a developmental program poised and then release it on schedule.

Krishnakumar et al., Cell Stem Cell 2016
2016

A miR-372/let-7 axis separates germ from somatic fate

In human cells, the balance between miR-372 and let-7 decides whether a cell becomes germline or soma, placing an ancient heterochronic circuit at a fate boundary.

Tran et al., Stem Cells 2016
2014

There is more than one route to pluripotency

The miR-290 and miR-302 clusters mark two distinct paths through late-stage reprogramming. Cells arriving at pluripotency have not necessarily taken the same road, which reframed reprogramming as a landscape rather than a single trajectory.

Parchem et al., Cell Stem Cell 2014
2013

MicroRNAs reveal the barriers to dedifferentiation

A microRNA-based screen identified the roadblocks that keep a fibroblast from returning to pluripotency, mapping the cellular checkpoints reprogramming must clear.

Judson et al., Nat Struct Mol Biol 2013

Cancer and developmental programs

2025 – 2021
2025

The same microRNA that patterns the brain also sizes the pancreas

miR-302 regulates the pancreatic progenitor pool and final organ size, extending its role in progenitor control well beyond the neural tube.

Yang et al., Biol Open 2025
2024

Cancers reactivate an embryonic enhancer landscape to stay addicted to SWI/SNF

Reactivation of the G1 enhancer landscape underlies core regulatory circuitry addiction to SWI/SNF, linking a chromatin remodeler's essentiality to a developmental program the tumor has reawakened.

Cermakova et al., Nucleic Acids Res 2024
2021

A chromatin assembly factor keeps neuroblastoma from growing up

CHAF1A blocks neuronal differentiation and drives neuroblastoma through metabolic reprogramming, showing a tumor holding itself in an immature developmental state.

Tao et al., Adv Sci (Weinh) 2021

Metabolism and neurodegeneration

2024 – 2022
2024

A blood-borne mutation and a bad diet compound each other

Hematologic DNMT3A reduction and high-fat diet synergize to promote weight gain and tissue inflammation, showing that a clonal hematopoiesis mutation and an environmental exposure are worse together than either alone.

Reyes et al., iScience 2024
2022

Antibodies against a transient toxic species extend survival in type 2 diabetes

hIAPP protofibrils are the toxic intermediate in type 2 diabetes, but they are transient and cannot be isolated, so they had never been drug targets. Working with collaborators at Rockefeller who locked hIAPP into the protofibril conformation, we showed these antibodies detect protofibrils in serum and islets without binding monomer, and that treating diabetic mice significantly extends survival. A species once considered undruggable is both a biomarker and a target.

Bortoletto et al., Adv Sci (Weinh) 2022

The maternal-fetal interface

2025 – 2018
2025

Amniotic fluid carries a readable record of fetal stress

Extracellular vesicles and cell-free RNA in amniotic fluid track how a fetus is adapting in twin-twin transfusion syndrome. The fluid around the fetus is not inert. It reports on the fetus inside it, which opens a route to monitoring a pregnancy without touching it.

Parchem et al., Am J Obstet Gynecol MFM 2025
2025

COVID-19 leaves a placental stress signature that mirrors preeclampsia

Single-nucleus profiling of placentas from patients with COVID-19 revealed ER stress, unfolded protein response activation, and CEBPB phosphorylation in the syncytiotrophoblast. The signature overlaps preeclampsia, offering a mechanism for a clinical association obstetricians had long observed. The samples came from Jackie’s patients and the profiling was done on Ron’s side. Neither half of the lab could have done it alone.

Keuls et al., Am J Obstet Gynecol 2025
2023

A fetus under cardiovascular stress rewires its metabolism

In twin-twin transfusion syndrome, twins share a placenta but not blood flow equally. Profiling that natural experiment showed how a fetal heart under load shifts its metabolism to cope, in a setting where the affected and unaffected twin share a genome.

Parchem et al., iScience 2023
2021

A preeclampsia risk gene acts through the kidney's blood pressure system

Loss of Stox1 in mice produces gestational hypertension driven by renin, alongside defects in the placenta itself. The result connects an inherited risk gene to a specific, and druggable, blood pressure pathway.

Parchem et al., JCI Insight 2021
2018

A preeclampsia biomarker turns out not to be a cause

Placental growth factor falls in preeclampsia and is used clinically to predict it, which invited the assumption that losing it drives the disease. In mice, deleting it did the opposite and maternal hypertension improved. A marker of the disease is not the same as its mechanism.

Parchem et al., J Clin Invest 2018 · Perinatal Research Society Prize
people
Ron & Jackie
Ron
Ronald J. Parchem, Ph.D.
Associate Professor, Department of Molecular and Cellular Biology
Baylor College of Medicine · Joint appointments in Neuroscience, the Center for Cell and Gene Therapy, the Dan L. Duncan Comprehensive Cancer Center, and the Stem Cells and Regenerative Medicine Center

A developmental biologist working on how cells decide what to become, and what happens when those decisions fail. His research spans birth defects, cancer, and metabolic disease, combining mouse genetics with single-cell genomics. B.S. and M.S. University of Illinois at Urbana-Champaign, Ph.D. UC Berkeley with Nipam Patel, postdoctoral fellowship UCSF with Robert Blelloch. Founded the lab at Baylor College of Medicine in 2015. His work is supported by the NIH, CPRIT, the V Foundation, the Lustgarten Foundation, and the Hydrocephalus Association.

Awards
  • CPRIT Scholar in Cancer Research · Cancer Prevention and Research Institute of Texas
  • V Scholar Award · The V Foundation for Cancer Research
  • Innovator Award · Hydrocephalus Association
  • Outstanding Course Award · Baylor College of Medicine
  • Outstanding Lecturer Award · Baylor College of Medicine
ronald.parchem@bcm.edu
Jackie
Jacqueline G. Parchem, MD
Associate Professor, Division of Maternal-Fetal Medicine, McGovern Medical School
UTHealth Houston · Adjunct in the Center for Precision Health, UTHealth, and Molecular and Cellular Biology, Baylor College of Medicine

A physician-scientist working on placental biology and the disorders of pregnancy, and what the maternal environment does to a developing embryo. Her research bridges the clinic and the bench, combining patient-derived tissue with cell culture and animal models. B.A. UC Berkeley, M.D. UCSF, OB/GYN residency UCSF, maternal-fetal medicine fellowship UCSF and Baylor College of Medicine, postdoctoral fellowship at MD Anderson with Raghu Kalluri. Co-director of the Mother Baby Biobank at UTHealth. She attends on labor and delivery at Memorial Hermann and Harris Health LBJ. Her work is supported by the NIH, the Doris Duke Foundation, the Burroughs Wellcome Fund, and the Preeclampsia Foundation.

Awards
  • Young Physician-Scientist Award · American Society for Clinical Investigation
  • Clinical Scientist Development Award · Doris Duke Foundation
  • Career Award for Medical Scientists · Burroughs Wellcome Fund
  • Peter J. Pappas Research Award · Preeclampsia Foundation
  • Dean's Teaching Excellence Award · McGovern Medical School, UTHealth Houston
Jacqueline.G.Parchem@uth.tmc.edu
Lab members
Meg
Margaret (Meg) Harrington
Graduate Student, Cancer & Cell Biology
Cancer stem cells, diabetes, and Alzheimer's disease.
B.A. Biology, Vassar College. Previously research technician at the University of Michigan developing brain tissue clearing and expansion protocols. NIH F31 fellow.
Dilean
Dilean J. Murillo González
Graduate Student, Neuroscience
Maternal diabetes, neural tube closure, and microRNA mechanisms.
B.S. Biological Sciences, UT El Paso. MCB Tsai Award.
Mara
Mara Cardenas
Graduate Student, DDMT
miRNA regulation of tumor-immune interactions in pancreatic cancer.
B.S. Neuroscience, UT Austin. Previously research technician at BCM Center for Cell and Gene Therapy developing immunotherapies for cancer.
Madison
Madison Vu
Research Assistant
Pancreatic tumorigenesis and preeclampsia with patient-derived samples.
B.S. UT Austin, 2024.
Lab group Lab Lab Lab Lab Lab
news
Recent
2026

Rachel Keuls, Lehmann Award

Selected as Ph.D. Class Speaker for Baylor College of Medicine commencement.

2025

Margaret Harrington, NIH F31 funded

Meg received an NIH Ruth L. Kirschstein Predoctoral Individual National Research Service Award (F31).

2025

Dilean Murillo, Ming-Jer Tsai Memorial Scholarship

Department of Molecular and Cellular Biology Tsai Award.

2025

Margaret Harrington, Best Speaker Award

Center for Cell and Gene Therapy Retreat.

2024

Rachel Keuls, Timothy M. George Memorial Award

International Conference on Neural Tube Defects. Rachel is the only trainee in the 25-year history of the conference to win this award at two consecutive meetings.

2023

Rachel Keuls, NIH F31 funded

Impact score: 10. Percentile: 1%.

2022

Rachel Keuls, Timothy M. George Memorial Award

International Conference on Neural Tube Defects.

2022

Angelina Bortoletto, Best Speaker Award

Center for Cell and Gene Therapy Retreat.

2020

Rachel Keuls, Poster Competition, Honorable Mention

Society for Developmental Biology.

former trainees
Where they went
Rachel A. Keuls, Ph.D.
Now a postdoctoral fellow in the lab of Huda Zoghbi, Texas Children’s Hospital.
Zoey Z. Yang, Ph.D.
Now a postdoctoral fellow in the lab of Tushar Desai, Stanford University.
Ivanshi Patel, Ph.D.
Now a postdoctoral fellow in the lab of Kara Marshall, Baylor College of Medicine.
Angelina S. Bortoletto, M.D., Ph.D.
Now a pathology resident at Brigham and Women’s Hospital, Harvard Medical School.
Akihiko Miyauchi, M.D., Ph.D.
Now Assistant Professor of Pediatrics, Jichi Medical University, Japan.
Karin Kojima, M.D., Ph.D.
Now Associate Professor of Pediatrics, Jichi Medical University, Japan.
Young Sun Oh, Ph.D.
Now CEO of TargetLink Therapeutics and Research Professor, Seoul National University.
join us
Themes we're looking for people to work within

We are recruiting graduate students, postdocs, and technicians. These are the areas we are building in. If one of them is the question you want to spend years on, write to us.

Developmental origins of birth defects

Neural tube closure, hydrocephalus, and the progenitor populations that build the early brain.

The maternal-fetal interface

Placental biology, preeclampsia, and how the maternal environment reaches the embryo.

Cancer and developmental programs

How tumors reawaken the regulatory programs that built the embryo, in pancreatic cancer and neuroblastoma.

Metabolism and neurodegeneration

Protein misfolding in type 2 diabetes, and why it raises the risk of dementia.

Chromatin and post-transcriptional control

The machinery that decides which genes are read, when, and in what order.

Stem cell identity and reprogramming

What commits a cell to a fate, and what allows it to go back.

how we work
mouse geneticsconditional and lineage-tracing allelesreporter micesingle-cell and single-nucleus genomicschromatin profilingRNA-protein interaction mappingmetabolomicshuman pluripotent stem cellsplacental and trophoblast modelspatient-derived tissueimaging and 3D reconstructioncomputational genomics
Mouse embryo, fluorescence microscopy, Parchem Lab
Neural tube cross-sections, fluorescence microscopy, Parchem Lab
Mouse brain scans, Parchem Lab
Mouse embryo, fluorescence microscopy, Parchem Lab
teaching
We teach
Graduate courses, Baylor College of Medicine
  • 2023–Topics in Stem Cell BiologyCourse Director
  • 2019–Evolutionary Mechanisms of DevelopmentCourse Director
  • 2015–Genetics for NeuroscienceCourse Director
  • 2015–DevelopmentCourse Director
  • 2019–Foundations A
  • 2016–Models of Development
  • 2015–Brain Cell Biology
Clinical and fellow teaching, UTHealth Houston
  • 2017–Maternal-Fetal Medicine fellow lecturesStillbirth, preeclampsia, the placenta, and research career development
  • 2017–OB/GYN resident lecturesStillbirth, preeclampsia, postpartum hemorrhage
  • 2017–Clinical supervisionUltrasound interpretation and consults for MFM fellows. High-risk obstetrics clinic, inpatient antepartum, and labor and delivery for residents.
selected talks
Where we've spoken
Ron
  • 2026Japanese Society of Child Neurology68th Annual Meeting, Utsunomiya, Japan · Keynote
  • 2025Dartmouth CollegeMolecular and Systems Biology
  • 2024Society for Craniofacial Genetics and Developmental BiologyKansas City
  • 2024International Conference on Neural Tube DefectsVancouver
  • 2022FASEB Folate, Vitamin B12, and One-Carbon MetabolismAsheville
  • 2019International Collaboration Forum of Human Gene TherapyJikei University, Tokyo · Keynote
  • 2019Gordon Research ConferenceNeural Crest and Cranial Placodes, Lucca, Italy
  • 2019Shanghai Children's HospitalShanghai
  • 2018Gordon Research ConferenceNeural Development, Newport
Jackie
  • 2026ZOOMing in on Preeclampsia ResearchInternational Society for the Study of Hypertension in Pregnancy and the Preeclampsia Foundation
  • 2025ENFOLD Sci-Art MeetingBurroughs Wellcome Fund, Durham
  • 2024American Gynecological & Obstetrical SocietyNashville
  • 2024Mount Sinai Inaugural Women's Health SymposiumNew York · Preeclampsia: 100 Years and 0 Cures
  • 2024University of MinnesotaOB/GYN Grand Rounds
  • 2022Northwestern MedicineMaternal-Fetal Medicine Division Seminar
  • 2022Society for Reproductive InvestigationDenver
  • 2020University of MichiganOB/GYN Grand Rounds
  • 2019Fudan UniversityShanghai
service
Beyond the lab
Ron
  • NIH study sections. Developmental Brain Disorders (DBD), Molecular Genetics and Genomics (MGG), and Pregnancy and Neonatology (PN).
  • Foundation review panels. The V Foundation for Cancer Research, the Lustgarten Foundation, and the Hydrocephalus Association.
  • Baylor College of Medicine. Chair of the Molecular and Cellular Biology faculty search, chair of the Stem Cells and Regenerative Medicine Center faculty search, and the DDMT graduate program admissions committee.
  • Community. Co-organizer of the MCB retreat, the Center for Cell and Gene Therapy retreat, and the junior faculty mentorship group.
Jackie
  • NIH study sections. Advancing Therapeutics B (ATB) and career development award panels. Member of the NICHD Human Placenta Project planning committee.
  • Editorial. Editorial board of Scientific Reports. Reviewer for AJOG, Obstetrics & Gynecology, eLife, JAMA Network Open, Hypertension, and others.
  • Society for Maternal-Fetal Medicine. Publications committee, communications committee vice chair, and co-lead of the President's Workshop on Preeclampsia.
  • National. Consulting member of the ASCI Physician-Scientist Development Committee. Engagement lead, Perinatal Research Society Council. Preeclampsia Foundation expert directory and grant review.
public scholarship
Talking to people who are not scientists

During the pandemic, pregnant patients were asked to make decisions about a vaccine that had been tested on almost no one like them. Jackie spent that period explaining what was known and what was not, to reporters, to patients, and to Congress.

Advocacy and education
  • Capitol Hill briefing. Speaker on behalf of the Society for Maternal-Fetal Medicine, with the NICHD and March of Dimes, on the imperative to include pregnant and lactating people in clinical trials.
  • ACOG Expert View. Patient education column on the questions she asked herself about the COVID-19 vaccine, while pregnant.
  • Preeclampsia Foundation. Ask the Experts Live panelist for Preeclampsia Awareness Month, and the #MakePreeclampsiaHistory campaign.
  • Society for Maternal-Fetal Medicine. Moderator and panelist, Myth-Busting COVID-19 Vaccines in Pregnancy.
Selected coverage
  • TIME. Why there are no data on COVID-19 vaccination and pregnancy. Jackie walked through her own decision to get vaccinated at 31 weeks.
  • Scientific American. Vaccination protects pregnant people and their babies from severe COVID. On what she was seeing on labor and delivery: “I was training when the H1N1 pandemic happened, and that was the most similar time that I can remember. This is an order of magnitude worse.”
  • USA TODAY. Pregnant women didn't have the data. Until now.
  • The Washington Post. They held out on getting the vaccine. This is what changed their minds.
  • MedPage Today. The onus is on us to get our pregnant patients vaccinated. Invited opinion.
TIMEScientific AmericanUSA TODAYThe Washington PostU.S. News & World ReportHouston ChronicleWebMDABC13KHOU11KPRCNBC4 WashingtonNew York PostWomen's HealthThe Fuller Project
funding

The lab is continuously funded by the NIH across both institutions, with additional support from foundations in cancer, birth defects, and maternal health.

National Institutes of Health
Cancer Prevention and Research Institute of Texas
The V Foundation for Cancer Research
Lustgarten Foundation
Hydrocephalus Association
Burroughs Wellcome Fund
Doris Duke Foundation
Preeclampsia Foundation
Robertson Foundation
Andrew McDonough B+ Foundation
Mike Hogg Foundation
Foundation for SMFM
papers
From the lab
miR-302 regulates pancreatic progenitor pool and pancreatic size
Biol Open, 2026 · read →
Placental dysfunction and congenital heart disease: investigating the placenta-heart axis
Prenatal Diagnosis, 2026 · in press
Opportunities and barriers for innovation at the interface of engineering and maternal-fetal medicine review
Commun Eng, 2026 · in press
Single-nucleus transcriptional profiling of the placenta reveals the syncytiotrophoblast stress response to COVID-19
Am J Obstet Gynecol, 2025 · read →
The miR-290 and miR-302 clusters are essential for reprogramming of fibroblasts to induced pluripotent stem cells
Stem Cells, 2025 · read →
Amniotic fluid extracellular vesicle and cell-free RNA profiling reveals fetal adaptations in twin-twin transfusion syndrome
Am J Obstet Gynecol MFM, 2025 · read →
COVID-19 infection history as a risk factor for early pregnancy loss
BMC Med, 2025 · read →
Hematologic DNMT3A reduction and high-fat diet synergize to promote weight gain and tissue inflammation
iScience, 2024 · read →
Reactivation of the G1 enhancer landscape underlies core circuitry addiction to SWI/SNF
Nucleic Acids Res, 2024 · read →
Post-transcriptional regulation in cranial neural crest cells expands developmental potential
PNAS, 2023 · read →
Fetal metabolic adaptations to cardiovascular stress in twin-twin transfusion syndrome
iScience, 2023 · read →
Racial and ethnic disparities in adverse perinatal outcomes at term
Am J Perinatol, 2023 · read →
Maternal metabolism influences neural tube closure review
Trends Endocrinol Metab, 2023 · read →
KRAS hijacks the miRNA regulatory pathway in cancer review
Cancer Res, 2023 · read →
The role of noncoding RNAs in pancreatic birth defects review
Birth Defects Res, 2023 · read →
A pancreatic player in dementia: pathological role for islet amyloid polypeptide accumulation in the brain review
Neural Regen Res, 2023 · read →
Aspirin discontinuation in pregnancies at high risk of preterm preeclampsia review
JAMA, 2023 · read →
Human islet amyloid polypeptide (hIAPP) protofibril-specific antibodies for detection and treatment of type 2 diabetes
Adv Sci (Weinh), 2022 · read →
Pregnancy and the risk of in-hospital COVID-19 mortality
Obstet Gynecol, 2022 · read →
Maternal diabetes negatively impacts fetal health review
Open Biol, 2022 · read →
Regulation of Oct4 in stem cells and neural crest cells review
Birth Defects Res, 2022 · read →
Preeclampsia: a report and recommendations of the workshop of the Society for Maternal-Fetal Medicine and the Preeclampsia Foundation review
Am J Obstet Gynecol, 2022 · read →
More than grit: growing and sustaining physician-scientists in obstetrics and gynecology review
Am J Obstet Gynecol, 2022 · read →
CHAF1A blocks neuronal differentiation and promotes neuroblastoma oncogenesis via metabolic reprogramming
Adv Sci (Weinh), 2021 · read →
Single-cell multiomic approaches reveal diverse labeling of the nervous system by common Cre-drivers
Front Cell Neurosci, 2021 · read →
Stox1 deficiency is associated with renin-mediated gestational hypertension and placental defects in mice
JCI Insight, 2021 · read →
The COVID-19 vaccine in pregnancy: risks, benefits and recommendations review
Am J Obstet Gynecol, 2021 · read →
miR-302 regulates glycolysis to control cell-cycle during neural tube closure
Int J Mol Sci, 2020 · read →
Preparation of small RNA libraries for sequencing from early mouse embryos
J Vis Exp, 2020 · read →
Disparities in adverse maternal outcomes among Asian women at term
JAMA Netw Open, 2020 · read →
Adverse infant and maternal outcomes among low-risk term pregnancies stratified by race and ethnicity
Obstet Gynecol, 2020 · read →
Placental growth factor and the risk of adverse neonatal and maternal outcomes
Obstet Gynecol, 2020 · read →
Conformation-specific antibodies against multiple amyloid protofibril species from a single amyloid immunogen
J Cell Mol Med, 2019 · read →
Loss of placental growth factor ameliorates maternal hypertension and preeclampsia in mice
J Clin Invest, 2018 · read →
Utility of chromosomal microarray in anomalous fetuses
Prenatal Diagnosis, 2018 · read →
A miR-372/let-7 axis regulates human germ versus somatic cell fates
Stem Cells, 2016 · read →
FOXD3 regulates pluripotent stem cell potential by simultaneously initiating and repressing enhancer activity
Cell Stem Cell, 2016 · read →
miR-302 is required for timing of neural differentiation, neural tube closure, and embryonic viability
Cell Reports, 2015 · read →
Two miRNA clusters reveal alternative paths in late-stage reprogramming
Cell Stem Cell, 2014 · read →
MicroRNA-based discovery of barriers to dedifferentiation of fibroblasts to pluripotent stem cells
Nat Struct Mol Biol, 2013 · read →
Analysis of snail genes in the crustacean Parhyale hawaiensis: insight into snail gene family evolution
Dev Genes Evol, 2012 · read →
A prominent requirement for single-minded and the ventral midline in patterning the dorsoventral axis of the crustacean Parhyale hawaiensis
Development, 2010 · read →
BAC library for the amphipod crustacean, Parhyale hawaiensis
Genomics, 2010 · read →
Patterns on the insect wing review
Curr Opin Genet Dev, 2007 · read →
Parchem Lab Parchem Lab Development & Disease
contact

Join us or get in touch

We're recruiting graduate students, postdocs, and technicians. Write to either of us.

Ron Parchem

ronald.parchem@bcm.edu

Room N1020, Baylor College of Medicine
One Baylor Plaza, Houston, TX 77030
Jackie Parchem

Jacqueline.G.Parchem@uth.tmc.edu

MSB 3.286, McGovern Medical School
6431 Fannin St, Houston, TX 77030
Texas Medical Center · Houston, Texas